Manchester Peptides catalogue profile
MOTS-C
A mitochondria-derived peptide studied as a signal connecting cellular stress, energy use and metabolic adaptation. Research explores mitochondrial function, insulin sensitivity and exercise-response pathways, although most intervention evidence remains preclinical.
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Prices shown are the current catalogue guide prices.Vial · 50 mg
MOTS-C
Product information
A mitochondria-derived peptide studied as a signal connecting cellular stress, energy use and metabolic adaptation. Research explores mitochondrial function, insulin sensitivity and exercise-response pathways, although most intervention evidence remains preclinical.
Evidence and sources at a glance
- Human evidence
- Human work includes observational biology and early experimental research, not an established therapeutic evidence base.
- Regulatory context
- MOTS-C is not an approved medicine for metabolism, exercise performance or ageing.
- Original MOTS-C mechanistic study ↗Primary 2015 study using cell and mouse models; it does not establish a treatment effect in humans.
- MOTS-C mechanisms review ↗Peer-reviewed review of mitochondrial and metabolic mechanisms.
Pen · 40 mg
MOTS-C
Product information
A mitochondria-derived peptide studied as a signal connecting cellular stress, energy use and metabolic adaptation. Research explores mitochondrial function, insulin sensitivity and exercise-response pathways, although most intervention evidence remains preclinical.
Evidence and sources at a glance
- Human evidence
- Human work includes observational biology and early experimental research, not an established therapeutic evidence base.
- Regulatory context
- MOTS-C is not an approved medicine for metabolism, exercise performance or ageing.
- Original MOTS-C mechanistic study ↗Primary 2015 study using cell and mouse models; it does not establish a treatment effect in humans.
- MOTS-C mechanisms review ↗Peer-reviewed review of mitochondrial and metabolic mechanisms.
Plain-English identity
What is MOTS-C?
Researchers investigate how MOTS-C relates to insulin sensitivity, metabolic flexibility, exercise response, inflammation, cellular stress and biological ageing.
Most intervention evidence remains preclinical. Human studies have explored associations and biological responses, but marketed research MOTS-C is not an approved treatment for performance, ageing or metabolic disease.
Evidence and sources at a glance
- Human evidence
- Human work includes observational biology and early experimental research, not an established therapeutic evidence base.
- Regulatory context
- MOTS-C is not an approved medicine for metabolism, exercise performance or ageing.
- Original MOTS-C mechanistic study ↗Primary 2015 study using cell and mouse models; it does not establish a treatment effect in humans.
- MOTS-C mechanisms review ↗Peer-reviewed review of mitochondrial and metabolic mechanisms.
Third-party documentation
Certificates of Analysis
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Browse all available COAsEvidence-led research guide
Go deeper into MOTS-C
A mitochondria-derived peptide studied as a signal connecting cellular stress, energy use and metabolic adaptation.
Last reviewed 10 October 2026. Research and reference information only. This page does not provide dosing instructions, treatment recommendations or medical advice. Not for human or veterinary consumption.